Key result
Continuing competitive sports in young athletes with low-complexity VPBs and no structural cardiomyopathy did not increase sudden death risk, with median daily VPBs decreasing from 93 to 72.
Why the study?
Does continuing competitive sports without detraining increase arrhythmic risk in young athletes with isolated ventricular premature beats?
Cohort (n=3,746)
Does continuing competitive sports without detraining increase arrhythmic risk in young athletes with isolated ventricular premature beats?
Continuing competitive sports in young athletes with frequent but low-complexity ventricular premature beats and no structural heart disease does not increase arrhythmic risk and is associated with a decrease in VPB burden.
Answers are grounded in this paper and cite their sources.
May support continued training without detraining in low-risk athletes with isolated VPBs; hypothesis-generating and requires prospective validation.
Isolated ventricular premature beats (VPBs) are commonly found during pre-participation screening in athletes. Currently, the debate about the role of detraining in reducing the number of VPBs is still open. This study evaluated the arrhythmic risk in a population of young competitive athletes who showed VPBs during eligibility evaluation and that did not undergo detraining but continued practicing competitive sports. 3746 consecutive subjects underwent pre-participation screening. Athletes who showed VPBs were selected and underwent second level evaluation (Echocardiogram, 24 hour Holter ECG and Exercise test). Athletes were re-evaluated after a follow-up period (6-48 months) while they continued practicing competitive sports. 5.3% of the whole population showed ventricular arrhythmias. 73% of the subjects showed isolated VPBs. 88% of the subjects showed monomorphic VPBs, and 12% of athletes showed polymorphic VPBs. At echocardiogram, there was not any pathology which contraindicated competitive sport activity. At 24 hour Holter ECG recording, mean number of daily VPBs was 1592±3217 (range 0-16678). At holter ECG follow-up (16±12 months), the median number of VPBs decreased from 93 (IQR 20-3065) to a new value of 72 (IQR 2-1299). Continuing competitive sport in subjects with ventricular arrhythmias even though frequent but with a low grade of complexity and without structural cardiomyopathy does not increase sudden death risk.
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Parisi et al. (2017) conducted a cohort in Ventricular premature beats (n=3,746). Continuing competitive sports was evaluated on Median number of daily VPBs. Continuing competitive sports in young athletes with low-complexity VPBs and no structural cardiomyopathy did not increase sudden death risk, with median daily VPBs decreasing from 93 to 72.
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