Correction to “Telomerase Knockout in Myeloid Cells Predisposes Mice to Foam Cell Formation, Dyslipidemia, Lung Fibrosis, and Cardiac Dysfunction”

Gao, Z., Y. Yu, D. Wiggins, E. M. Sevick-Muraca, and M. G. Kolonin. 2026. “Telomerase Knockout in Myeloid Cells Predisposes Mice to Foam Cell Formation, Dyslipidemia, Lung Fibrosis, and Cardiac Dysfunction.” Aging Cell 25: e70490. https://doi.org/10.1111/acel.70490. FIGURE 4. AT abnormalities in LysM-Tert KO mice. (a) Senescence-associated β-galactosidase staining of VAT from 20-month-old female mice. (b) Senescence-associated β-galactosidase staining of adherent cells from VAT in (a). Arrows: Senescent cells. (c) Flow cytometry on VAT from A, revealing a lower frequency of mG+ macrophages expressing CD206 in KO mice. (d) Flow cytometry on SAT, revealing a higher frequency of mG+ macrophages expressing CD86 in KO mice. (e) IF with antibodies against CD68 and CD206 reveals a lower frequency of CD206+ macrophages (red arrows) in SAT of KO mice. IF with antibodies against perilipin-1 and F4/80 reveals comparable adipocyte size in SAT of WT and KO mice. (f) Trichrome staining reveals fibrosis (arrows) in SAT of KO mice. In (d, e) 4-month-old male mice fed an atherogenic diet were used. Scale bar: 50 μm. We apologize for this error.