Association of Type D Personality and Anatomical Complexity as Predictors of Long-Term Mortality in Coronary Artery Disease: A Retrospective Case Study Based on Hospital Records

Figures

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  Figure 1. (Boxplots): Distribution of SCL-90 scores by personality type. The boxplots visually confirm the significantly higher psychometric distress symptom severity in Type D patients across all analyzed domains (Depression, Anxiety, and Hostility).

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  Figure 2. (Bar Chart): Mortality rate comparison (%) between groups. Although the Non-Type D group shows a higher raw percentage of events (34.6% vs. 26.2%), the difference is not statistically significant in this snapshot.

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  Figure 3. (Kaplan–Meier): Survival probability over time (days of follow-up). The Log-Rank test (p = 0.11) shows a borderline trend, suggesting a potential survival difference that might reach significance in a larger cohort or with longer follow-up.

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  Figure 4. Forest Plot for Model 1 (Trait/Anatomical dimensions). None of the variables, including Syntax Score and Social Inhibition, reached statistical significance, although Social Inhibition showed a borderline trend (p=0.068).

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  Figure 5. Forest Plot for Model 2 (Psychological SCL-90 continuous domains). Anxiety symptom severity presented an exploratory baseline association with survival (p=0.049), with a Hazard Ratio of 0.94, suggesting a potential, hypothesis-generating inverse trend in this cohort. * indicates statistical significance at p < 0.05.

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  Figure 6. Heatmap of the correlation matrix for psychological and clinical variables. The color intensity and numerical values represent the strength of Pearson’s correlation, showing consistent psychological distress patterns across the Type D construct.

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  Figure 7. The Anxiety Paradox and Pathophysiological Mechanisms in Coronary Artery Disease. The diagram illustrates the dual impact of anxiety on coronary health. The physiological pathway shows how chronic stress triggers the hypothalamic–pituitary–adrenal (HPA) axis, increasing cortisol and catecholamines, which lead to endothelial dysfunction and oxidative stress. Conversely, the behavioral pathway (the “Anxiety Paradox”) suggests that moderate anxiety promotes proactive vigilance and superior treatment adherence. The final coronary outcome depends on the balance between these two forces, highlighting the importance of primary prevention through combined CCTA and psychological profiling [30].