The Dual Impact of Mobile Social Media Breaks on Cortical Arousal and Neurophysiological Recovery: A Spectral Analysis of EEG Signals

Arousal is essential for cognitive awareness, and it typically declines during prolonged tasks. Currently, mobile devices are frequently used during breaks to relax and counteract this decline. However, their impact on neurophysiological recovery remains poorly understood. Therefore, this study compared the effects of traditional quiet rest versus a mobile task break on cortical arousal using spectral analysis of electroencephalography (EEG) signals in twenty healthy young females (20–25 years). Raw EEG data were transformed using Fast Fourier Transform (FFT) to determine power spectral densities, with a state of physiological underarousal first induced via a prolonged eyes-closed condition. Results revealed that this state was characterized by reduced alpha/beta power and delta/theta synchronization starting at the 4th minute. Although traditional quiet rest suppressed delta/theta synchronization, it failed to sustain cortical arousal, with alpha and beta powers declining by the 8th minute. In contrast, passive social media browsing acted as a potent neurocognitive stimulant, not only sustaining arousal but markedly increasing high-frequency beta power by the 16th minute. Furthermore, preliminary network-level connectivity analysis using Phase Locking Value (PLV) revealed that mobile tasks induced widespread beta-band synchronization across frontal-midline regions, suggesting enhanced functional coupling within the executive control network. In conclusion, in healthy young females, while mobile tasks strategically counteract low arousal, they fail to facilitate the neurophysiological disengagement necessary for true recovery. These findings underscore the importance of digital hygiene, highlighting a distinction between alertness-boosting activities and recovery-focused rest. The results suggest that mobile tasks may create a subjective perception of rest despite objective signs of sustained cortical activation, implying that such activities may not facilitate genuine neurophysiological recovery within the context of short-term neurophysiological modulation.