Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
October 1, 2001Journal of Biological ChemistryOpen Access

ERK2- and p90Rsk2-dependent Pathways Regulate the CCAAT/Enhancer-binding Protein-β Interaction with Serum Response Factor

View Full Paper
Ask AI
Bookmark
Share

Authors

MHMary H. HanlonUniversity of MinnesotaTSThomas W. SturgillUniversity of VirginiaLSLinda SealyVanderbilt University

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Hanlon et al. (2001) studied this question.

synapsesocial.com/papers/6a5de9f2dec751667c70971ahttps://doi.org/10.1074/jbc.m102165200
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Ras Regulates the Association of Serum Response Factor and CCAAT/Enhancer-binding Protein β1999 · 38 citations
  2. 2C/EBPBeta and Elk-1 synergistically transactivate the c-fos serum response element2000 · 28 citations
  3. 3Ribosomal subunit kinase-2 is required for growth factor-stimulated transcription of the c- Fos gene2000 · 76 citations
  4. 4Activation of Raf as a Result of Recruitment to the Plasma Membrane1994 · 987 citations
  5. 5Novel Membrane-Targeted ERK1 and ERK2 Chimeras Which Act as Dominant Negative, Isotype-Specific Mitogen-Activated Protein Kinase Inhibitors of Ras-Raf-Mediated Transcriptional Activation of c- fos in NIH 3T3 Cells1999 · 39 citations