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November 1, 2002The Plant JournalOpen Access

The plant disease resistance gene Asc‐1 prevents disruption of sphingolipid metabolism during AAL‐toxin‐induced programmed cell death

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Authors

SSStefka D. SpassievaUniversity of KentuckyJMJonathan E. MarkhamUniversity of Nebraska–LincolnJHJacques HilleUniversity of Toronto

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Spassieva et al. (2002) studied this question.

synapsesocial.com/papers/6a653c9475f8bd916cc7f14fhttps://doi.org/10.1046/j.1365-313x.2002.01444.x
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Overexpression of the Tomato Asc-1 Gene Mediates High Insensitivity to AAL Toxins and Fumonisin B1 in Tomato Hairy Roots and Confers Resistance to Alternaria alternata f. sp. lycopersici in Nicotiana umbratica Plants2002 · 61 citations
  2. 2TRIPLES: a database of gene function in Saccharomyces cerevisiae2000 · 80 citations
  3. 3Accumulation of Phosphorylated Sphingoid Long Chain Bases Results in Cell Growth Inhibition in Saccharomyces cerevisiae2000 · 107 citations
  4. 4Involvement of Yeast Sphingolipids in the Heat Stress Response of Saccharomyces cerevisiae1997 · 302 citations
  5. 5Fumonisin B1 induces apoptosis in cultured human keratinocytes through sphinganine accumulation and ceramide depletion.1999 · 69 citations