PulseExploreJournal ClubDebatesTrendingResearchersJournals
Instagram
HomeExploreJournal ClubTrending
Synapse
⌘+K
Synapse
September 10, 2025Arthroscopy The Journal of Arthroscopic and Related Surgery0 citations

High-Dose Statins Preserve Tendon-Bone Interface Healing Without Adversely Affecting Fatty Infiltration in a Rotator Cuff Repair Rat Model

View Full Paper

Why the study?

Does high-dose atorvastatin affect tendon-bone interface healing and fatty infiltration following rotator cuff repair in a rat model?

Population

20 Sprague-Dawley rats undergoing rotator cuff repair surgery on both shoulders

Design

Preclinical

Follow-up

4 weeks

Key result

High-dose atorvastatin did not impair histologic and biomechanical healing of the tendon-bone interface and significantly inhibited the expression of the muscle atrophy marker MuRF-1 (P < .001).

Authors

JYJong Pil YoonSPSung-Jin ParkDKDong‐Hyun Kim

Discussion

Loading...

Member takes

Overview

May support statin continuation around rotator cuff repair; leaves open human translation.

Structured PICO

Does high-dose atorvastatin affect tendon-bone interface healing and fatty infiltration following rotator cuff repair in a rat model?

P
Population
20 Sprague-Dawley rats undergoing bilateral rotator cuff repair surgery, followed for 4 weeks.
I
Intervention
High-dose atorvastatin administered orally for 4 weeks postoperatively
O
Outcome
Histologic and biomechanical tendon-bone interface (TBI) healing and fatty infiltrationsurrogate

High-dose atorvastatin does not negatively affect tendon-bone interface healing or muscle recovery following rotator cuff repair in a rat model, supporting its continued use in patients requiring statin therapy.

Ask AI
Helpful
Bookmark
Share
View Full Paper

Cite This Study

Yoon et al. (2025) studied Rotator cuff repair (n=20). High-dose atorvastatin vs. Control was evaluated on Histologic and biomechanical tendon-bone interface healing and fatty infiltration. High-dose atorvastatin did not impair histologic and biomechanical healing of the tendon-bone interface and significantly inhibited the expression of the muscle atrophy marker MuRF-1 (P < .001).

synapsesocial.com/papers/6a6a17ea02245592faf1944dhttps://doi.org/10.1016/j.arthro.2025.07.043
Ask AI
Helpful
Bookmark
Share
View Full Paper