In Vivo Recruitment of XPC to UV-induced Cyclobutane Pyrimidine Dimers by the DDB2 Gene Product

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1. 1p53 responsive nucleotide excision repair gene products p48 and XPC, but not p53, localize to sites of UV-irradiation-induced DNA damage, in vivo2003 · 80 citations
2. 2Characterization of a human DNA damage binding protein implicated in xeroderma pigmentosum E.1993 · 212 citations
3. 3DDB Accumulates at DNA Damage Sites Immediately after UV Irradiation and Directly Stimulates Nucleotide Excision Repair2002 · 228 citations
4. 4Sequential binding of UV DNA damage binding factor and degradation of the p48 subunit as early events after UV irradiation2002 · 173 citations
5. 5Cyclobutane Pyrimidine Dimers Are Responsible for the Vast Majority of Mutations Induced by UVB Irradiation in Mammalian Cells2001 · 318 citations