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TOXOPLASMOSIS as a congenital infection is now a well-established clinical entity, but our knowledge of toxoplasmosis acquired in childhood or adult life is much less complete. It is known, however, that such infections may occur, and several cases without ocular involvement have been reported (Pinkerton and Most of the cases of acquired toxoplasmosis in man have not shown ocular involvement, and Sabin (1950) and Sabin and others (1952), have emphasized the fact that there is as yet no satisfactory evidence that the organism can cause a chorio-retinitis through a post-natally acquired infec- tion. Although he did not deny its possible occurrence, Sabin (1950) pointed out that the fully developed and post-natal nervous tissues are un- questionably less susceptible to the pathogenic effects of toxoplasma than are those of the embryonic nervous system.
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Duke-Elder et al. (1953) studied this question.
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