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Extends biochemical insights into metachromatic leukoencephalopathy; leaves open genetic mechanisms and therapeutic targets.
THE delineation by Brain and Greenfield (1950) of a late infantile form of metachromatic leuco-encephalopathy has been followed by the im-portant discovery that the brain in this condition contains a large increase in sulphatide (Jatzkewitz, 1958; Austin, 1959). Our study of two familial
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Norman et al. (1960) studied this question.
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