In an animal model of hyperlipoproteinemia type I, LPL deficiency leads to retarded chylomicron clearance and abnormal hepatic uptake of triglycerides instead of normal peripheral tissue distribution.
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LPL deficiency redirects chylomicron triglycerides to liver in mink; hypothesis-generating for type I hyperlipoproteinemia mechanisms and requires human confirmation.
Savonen et al. (1999) studied this question.
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