Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
October 31, 2006Journal of Biological ChemistryOpen Access

Orphan Nuclear Receptor Small Heterodimer Partner Inhibits Transforming Growth Factor-β Signaling by Repressing SMAD3 Transactivation

View Full Paper
Ask AI
Bookmark
Share

Authors

JSJi Ho SuhThe University of Texas Health Science Center at HoustonJHJiansheng HuangAnyang Academy of Agricultural SciencesYPYun‐Yong ParkChung-Ang University

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Suh et al. (2006) studied this question.

synapsesocial.com/papers/6a703508fe4101aa97e00dc6https://doi.org/10.1074/jbc.m605947200
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Orphan Nuclear Receptor Small Heterodimer Partner Represses Hepatocyte Nuclear Factor 3/Foxa Transactivation via Inhibition of Its DNA Binding2004 · 83 citations
  2. 2Differential Role of the Loop Region between Helices H6 and H7 within the Orphan Nuclear Receptors Small Heterodimer Partner and DAX-12004 · 31 citations
  3. 3Estrogen Receptor α Regulates Expression of the Orphan Receptor Small Heterodimer Partner2003 · 80 citations
  4. 4The Small Heterodimer Partner Interacts with the Pregnane X Receptor and Represses Its Transcriptional Activity2003 · 114 citations
  5. 5SMAD Proteins Transactivate the Human ApoCIII Promoter by Interacting Physically and Functionally with Hepatocyte Nuclear Factor 42000 · 42 citations