ANG II-induced mesenteric vasoconstriction is mediated by intracellular Ca2+ and PKC, whereas TxA2-induced vasoconstriction utilizes different, unidentified signaling pathways.
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Supports pathway-specific mesenteric vasoconstriction in animals; leaves open TxA2 targets and human translation.
Bauer et al. (1999) studied this question.
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