Key result
Toll-like receptor signaling links autoimmune diseases to accelerated atherosclerosis.
Key points are not available for this paper at this time.
Review examines the potential role of signaling by endogenous TLR ligands in the pathogenesis of autoimmune disease and atherosclerosis and explores how they might be related.
No takes yet. Share an insight, caveat, or question.
Huang et al. (2010) studied this question. Toll-like receptor signaling by endogenous ligands acts as a potential pathogenetic link between autoimmune diseases, including rheumatoid arthritis and systemic lupus, and atherosclerosis.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: