noted for some time, especially in the conditions of hyperparathyroidism (Cogan, Albright, and Bartter, 1948; More recently Berlyne and Shaw (I967, I968) reported similar deposits in patients with acute and chronic renal failure in whom by contrast there was hypocalcaemia and marked hyperphosphataemia. They also found that the calcium and inorganic phosphate product was usually greater than 70 in these patients, and further suggested that these deposits were responsible for the red eye of renal failure. Other workers have confirmed the presence of these deposits in uraemic patients but have questioned the aetiology of the red eye (Harris, Cohn, Toyofuku, Lonergan, and Galin, I97I; Caldeira, Sabbaga, and Ianhez, I970).
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Porter et al. (1973) studied this question.
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