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March 1, 1994Journal of Biological ChemistryOpen Access

Cyclic AMP-dependent phosphorylation of an immunoaffinity-purified homotetrameric inositol 1,4,5-trisphosphate receptor (type I) increases Ca2+ flux in reconstituted lipid vesicles.

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Authors

SNShinji NakadeKansai Medical UniversitySRSang Ki RheeSungkyunkwan UniversityHHHiroki HamanakaRIKEN Center for Brain Science

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Cite This Study

Nakade et al. (1994) studied this question.

synapsesocial.com/papers/6a70c8c82fdcb5703edb6830https://doi.org/10.1016/s0021-9258(17)37437-9
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The effect of external calcium and pH on inositol trisphosphate-mediated calcium release from cerebellum microsomal fractions1989 · 140 citations
  2. 2Immunohistochemical localization of an inositol 1,4,5-trisphosphate receptor, P400, in neural tissue: studies in developing and adult mouse brain1991 · 125 citations
  3. 3Involvement of the <i>C</i>-terminus of the inositol 1,4,5-trisphosphate receptor in Ca2+ release analysed using region-specific monoclonal antibodies1991 · 95 citations
  4. 4Sequence and functional characterization of a third inositol trisphosphate receptor subtype, IP3R-3, expressed in pancreatic islets, kidney, gastrointestinal tract, and other tissues1993 · 319 citations
  5. 5The subtypes of the mouse inositol 1,4,5-trisphosphate receptor are expressed in a tissue-specific and developmentally specific manner.1991 · 266 citations