Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
August 1, 2002BloodOpen Access

von Willebrand factor cleaving protease (ADAMTS13) is deficient in recurrent and familial thrombotic thrombocytopenic purpura and hemolytic uremic syndrome

View Full Paper
Ask AI
Bookmark
Share

Authors

Giuseppe Remuzzi
Giuseppe RemuzziUniversity of BergamoMGMiriam GalbuseraMario Negri Institute for Pharmacological ResearchMNMarina NorisMario Negri Institute for Pharmacological Research

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Remuzzi et al. (2002) studied this question.

synapsesocial.com/papers/6a7104ff2163a0a01bc53600https://doi.org/10.1182/blood-2001-12-0166
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Specific von Willebrand factor–cleaving protease in thrombotic microangiopathies: a study of 111 cases2001 · 384 citations
  2. 2Enhanced proteolysis of plasma von Willebrand factor in thrombotic thrombocytopenic purpura and the hemolytic uremic syndrome1989 · 60 citations
  3. 3Estimation of the von Willebrand Factor-cleaving Protease in Plasma Using Monoclonal Antibodies to vWF1999 · 112 citations
  4. 4Hereditary Thrombotic Thrombocytopenic Purpura: Microangiopathic Hemolytic Anemia, Thrombocytopenia, and Renal Insufficiency Occurring in Consecutive Generations2008 · 21 citations
  5. 5Bleeding in renal failure: is von Willebrand factor implicated?1977 · 58 citations