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July 15, 1999Blood

Increased Fragmentation of von Willebrand Factor, Due to Abnormal Cleavage of the Subunit, Parallels Disease Activity in Recurrent Hemolytic Uremic Syndrome and Thrombotic Thrombocytopenic Purpura and Discloses Predisposition in Families

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Authors

MGMiriam GalbuseraMario Negri Institute for Pharmacological ResearchMNMarina NorisMario Negri Institute for Pharmacological ResearchCRChiara RossiUniversity of Pisa

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Cite This Study

Galbusera et al. (1999) studied this question.

synapsesocial.com/papers/6a7104ff2163a0a01bc53602https://doi.org/10.1182/blood.v94.2.610
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Also Consider

Synapse has enriched 2 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Partial purification and characterization of a protease from human plasma cleaving von Willebrand factor to fragments produced by in vivo proteolysis1996 · 904 citations
  2. 2Heterogeneity of plasma von Willebrand factor multimers resulting from proteolysis of the constituent subunit.1991 · 198 citations