Lesions of the lateral parolivary area of the medulla oblongata, such as occur in the syndromes described by Avellis,¹Wallenberg,²Babinski and Nageotte³and Cestan and Chenais,⁴were after Wallenberg's⁵work generally regarded as due to occlusion of the posterior inferior cerebellar artery. Breuer and Marburg,⁶however, pointed out that similar lesions may be produced by thrombosis of the vertebral artery distal to the point of origin of the posterior inferior cerebellar artery or by that of the basilar artery, a fact which was confirmed by the extensive studies of Stopford.⁷In 1925 Foix, Hillemand and Schalit⁸described a new artery, the arteria fossae lateralis bulbi, and brought substantial evidence to demonstrate that most lesions of the lateral parolivary area (Ziehen⁹), including part of the dorsal lamella of the inferior olive and the descending root of the trigeminus nerve and its
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Leo Alexander (1937) studied this question.
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