Why the study?
Does resveratrol reduce endothelial cell migration and monocytic cell chemotaxis in vitro?
Population
Human umbilical vein endothelial cells (HUVECs) and THP-1 monocytic cells
Comparison
Resveratrol (1-20 µmol/L) in vitro vs Untreated or ethanol-treated control cells
Design
Preclinical
Follow-up
up to 27 hours
Key result
Resveratrol dose-dependently inhibited spontaneous endothelial cell migration and MCP-1-induced monocytic cell chemotaxis in vitro.
Authors
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Hypothesis-generating for resveratrol in atherosclerosis; clinical translation untested and should not change practice.
Does resveratrol reduce endothelial cell migration and monocytic cell chemotaxis in vitro?
Resveratrol dose-dependently inhibits endothelial cell migration and MCP-1-induced monocytic cell chemotaxis in vitro, highlighting a potential mechanism for its cardioprotective and anti-atherosclerotic effects.
p-value: p=<0.05
Cicha et al. (2011) studied Atherosclerosis (in vitro models). Resveratrol vs. Control (untreated or ethanol-treated) was evaluated on Endothelial cell migration and MCP-1-induced monocytic cell chemotaxis (p=<0.05). Resveratrol dose-dependently inhibited spontaneous endothelial cell migration and MCP-1-induced monocytic cell chemotaxis in vitro.