The phenol‐degrading system which can be induced in Rhodococcus spec. P 1 brings about the metabolization of phenol via the β‐ketoadipate path (ortho‐cleavage of catechol). For the more precise characterization of the degradation performance investigations were made into the influences of different carbon sources: glucose, galactose, ribose, arabinose, xylose, acetate, citricacid, mandelic acid, protocatechoic acid, glycerol, tryptophane, phenylalanine and the complex substrate of nutrient broth as well as succinate, catechol and benzoate. Only the three last‐named substances show an influence: succinate inhibits only the synthesis of phenol hydroxylase, catechol and benzoate inhibit the synthesis and activity of phenol hydroxylase. As a comparison of the results with literature shows, even at the same path of degradation there are different regulating mechanismus in the different microorganisms.
No takes yet. Share an insight, caveat, or question.
Hensel et al. (1984) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: