Population
50 patients at risk for clinical acute respiratory failure (ARDS)
Design
Cohort
Follow-up
up to 10 days
Authors
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Elevated C3a and granulocyte aggregation associate with ARDS; supports complement activation hypothesis but leaves causality and therapeutic implications open.
Increased plasma C3a and granulocyte aggregation in patients developing ARDS suggest systemic complement activation is involved in its pathogenesis.
Gus J. Slotman (1986) studied this question.
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