Why the study?
Does unilateral lung ischemia generate interleukin-8-mediated chemotactic signals for PMN accumulation in a sheep model?
Does unilateral lung ischemia generate interleukin-8-mediated chemotactic signals for PMN accumulation in a sheep model?
Unilateral lung ischemia in sheep generates an interleukin-8-like chemotactic substance that may mediate polymorphonuclear leukocyte inflammation and post-ischemic injury.
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IL-8-like chemotaxis after lung ischemia may mediate PMN inflammation in sheep; leaves open relevance to human post-ischemic injury.
Wickersham et al. (1993) studied this question.
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