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June 1, 2000Journal of Molecular EndocrinologyOpen Access

Transcriptional activation of transforming growth factor alpha by estradiol: requirement for both a GC-rich site and an estrogen response element half-site

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Authors

CVCarrie A. VyhlidalUniversity of North Carolina at Chapel HillISIsmael SamudioFPInnovationsMKMichael P. KladdeUniversity of Florida Health

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Vyhlidal et al. (2000) studied this question.

synapsesocial.com/papers/6a73322f64decaab472c1d8ahttps://doi.org/10.1677/jme.0.0240329
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Cathepsin D gene is controlled by a mixed promoter, and estrogens stimulate only TATA-dependent transcription in breast cancer cells.1993 · 150 citations
  2. 2The Effects of a Constitutive Expression of Transforming Growth Factor-α on the Growth of MCF-7 Human Breast Cancer Cellsin Vitroandin Vivo1989 · 127 citations
  3. 3Estrogen Induces Adenosine Deaminase Gene Expression in MCF-7 Human Breast Cancer Cells: Role of Estrogen Receptor-Sp1 Interactions**This work was supported by the National Institutes of Health (Grant CA-76636), the Welch Foundation, and the Texas Agricultural Experiment Station.1999 · 83 citations
  4. 4Transcriptional Activation of E2F1 Gene Expression by 17β-Estradiol in MCF-7 Cells Is Regulated by NF-Y-Sp1/Estrogen Receptor Interactions1999 · 175 citations
  5. 5Expression of transforming growth factor α antisense mRNA inhibits the estrogen‐induced production of TGFα and estrogen‐induced proliferation of estrogen‐responsive human breast cancer cells1993 · 54 citations