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November 18, 2019Translational PsychiatryOpen Access

Mitochondrial deficits in human iPSC-derived neurons from patients with 22q11.2 deletion syndrome and schizophrenia

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Authors

JLJianping LiShenzhen Institutes of Advanced TechnologySRSean K. RyanAVEO Oncology (United States)EDErik DeBoerBristol-Myers Squibb (United States)

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Li et al. (2019) studied this question.

synapsesocial.com/papers/6a737fc31aa2fc9e78fb671dhttps://doi.org/10.1038/s41398-019-0643-y
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The 22q11 deletion syndromes2000 · 500 citations
  2. 2MLPA: a rapid, reliable, and sensitive method for detection and analysis of abnormalities of 22q2006 · 140 citations
  3. 3Schizophrenia1994 · 516 citations
  4. 4Tetracycline Antibiotics Impair Mitochondrial Function and Its Experimental Use Confounds Research2015 · 149 citations
  5. 5In the line-up: deleted genes associated with DiGeorge/22q11.2 deletion syndrome: are they all suspects?2019 · 86 citations