Key result
Circulating immune cells drive ischemic stroke pathogenesis by promoting immunothrombosis and plaque instability.
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This review highlights the emerging role of immune cells in driving immunothrombosis and thromboinflammation in acute ischemic stroke, which impacts vessel recanalization and patient outcomes.
Ischemic stroke is one of the leading causes of disability and mortality worldwide. Thrombosis is the main pathological process of stroke and is therefore an important therapeutic target in stroke prevention. In recent years, with the development of endovascular treatment and therefore retrieving the thrombus for further investigation, evidence is accumulating that immune cells are inextricably linked to stroke pathogenesis. Circulating immune cells have been found to induce immunothrombosis, and they actively participate in the formation of the thrombus by promoting platelet recruitment and thrombin activation. Additionally, the formation of thromboinflammation leads to increased instability of atherosclerotic plaques. We review the concepts of stroke immunothrombosis and thromboinflammation and the effect of immune cells on vessel recanalization and patient outcome. In addition, we elaborate on the possible mechanism of immune cells being activated and participating in thrombosis in ischemic stroke.
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Wang et al. (2024) conducted a review in Acute Ischemic Stroke. Immunothrombosis and thromboinflammation was evaluated. Circulating immune cells actively participate in ischemic stroke pathogenesis by inducing immunothrombosis, promoting platelet recruitment, and increasing atherosclerotic plaque instability.
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