Key result
Dipyridamole-induced stress demonstrated normalization of left ventricular rigid body rotation at maximum hyperaemia in a patient with stable angina.
Why the study?
Does dipyridamole-induced stress reverse left ventricular rigid body rotation in a patient with stable angina?
Case Report (n=1)
Does dipyridamole-induced stress reverse left ventricular rigid body rotation in a patient with stable angina?
Dipyridamole-induced stress can normalize left ventricular rigid body rotation in patients with stable angina, as demonstrated by 3D speckle tracking echocardiography.
May indicate reversible mechanics in stable angina; leaves open prospective validation before any clinical role.
The left ventricular (LV) twist is defined as the wringing motion of the heart around its long-axis in systole caused by oppositely directed counterclockwise apical and clockwise basal rotations resulted from the movement of two orthogonally oriented muscular bands. In some clinical circumstances, rotation at both basal and apical levels of the LV occurred in the same clockwise or counterclockwise direction during systole resulting the near absence of LV twist as called left ventricular "rigid body rotation" (LV-RBR). Hereby we present that LV-RBR normalization of LV rotational mechanics could be demonstrated at maximum hyperaemia during dipyridamole-induced stress by three-dimensional (3D) speckle tracking echocardiography in a patient with stable angina.
No takes yet. Share an insight, caveat, or question.
Nemes et al. (2016) conducted a case report in stable angina (n=1). dipyridamole-induced stress was evaluated on normalization of left ventricular rotational mechanics. Dipyridamole-induced stress demonstrated normalization of left ventricular rigid body rotation at maximum hyperaemia in a patient with stable angina.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: