CASES of toxicity from ferrous sulfate have been reported sporadically for two centuries. Revival of interest in acute iron poisoning stems back, however, to the report of Forbes1 in 1947, which included a description of clinical symptoms, pathological findings and an experimental study demonstrating the toxicity of large doses of iron compound. Since that time, many reports of both fatal and nonfatal poisoning have appeared, nearly all of which followed the accidental ingestion of ferrous sulfate tablets by children. In addition interest in the role of iron and its compounds and metabolites in human siderosis, hemosiderosis and cirrhosis of the . . .
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Luongo et al. (1954) studied this question.
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