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Introduction Morphological and cytogenetic features categorize myeloid neoplasms into myelodysplastic syndromes (MDS), myelodysplastic/myeloproliferative neoplasms (MDS/MPN), myeloproliferative neoplasms (MPN) and AML [1]. Specific genetic abnormalities are identified in some subtypes of these diseases. These abnormalities frequently target transcriptional factors involved in controlling cell proliferation and differentiation and are associated with different clinical
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Koduru et al. (2016) studied this question.
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