UNILATERAL renal disease, as a remediable cause of hypertension, has attracted much clinical interest since the suggestive studies of Goldblatt et al.1 in 1934. Since the initial clinical report by Butler2 in 1937, numerous patients with various forms of unilateral renal disease have undergone nephrectomy in an attempt to relieve hypertension. long-term results in the larger series, however, have not been impressive, the cure rate ranging from 19 to 24 per cent.3 , 4 In recent years, notably since the advent of translumbar aortography, attention has focused on lesions involving the renal vasculature, and it is in this group that a reasonable . . .
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Vincent J. O’Conor (1960) studied this question.
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