Membrane depolarization inhibits Kv1.5 gene transcription and protein expression in pituitary cells, which may produce a long-term enhancement of cell excitability.
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Depolarization may enhance pituitary cell excitability via Kv1.5 suppression; leaves open relevance to cardiac remodeling or arrhythmias in humans.
Levitan et al. (1995) studied this question.
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