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Highlights the emerging evidence linking inflammation, specifically C-reactive protein levels, to atrial structural remodeling and outcomes after cardioversion in atrial fibrillation.
We read with considerable interest the article by Engelmann and Svendsen1 providing a concise overview of the current knowledge about the association of inflammation with atrial fibrillation (AF). A continuously increasing number of investigations examine the role of inflammation in AF. Interestingly, two recent studies indicated that C-reactive protein relates to the left atrial size and AF duration before cardioversion, providing evidence of an association between inflammation and atrial structural remodelling.2,3 Moreover, it has been demonstrated that baseline C-reactive protein levels prior cardioversion of persistent AF represent an independent predictor of sinus rhythm maintenance …
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Panagiotis Korantzopoulos (2005) studied this question.
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