It is argued that the static adult neuropsychological model is inappropriate for explaining the development of many atypical children whose brains cannot be considered in terms of a normal brain with parts intact and parts impaired. Rather, their brains develop differently from normal children from the very outset. I illustrate this with two examples from Williams syndrome, showing how equivalent behavioral outcomes stem from different underlying processes.
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Annette Karmiloff‐Smith (1997) studied this question.
Synapse has enriched 2 closely related papers on similar clinical questions. Consider them for comparative context: