Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
December 1, 2006The Journal of ImmunologyOpen Access

Interleukin-23 Restores Immunity to Mycobacterium tuberculosis Infection in IL-12p40-Deficient Mice and Is Not Required for the Development of IL-17-Secreting T Cell Responses

View Full Paper
Ask AI
Bookmark
Share

Authors

TWTeresa M. WozniakCommonwealth Scientific and Industrial Research OrganisationARAnthony A. RyanThe University of SydneyWBWarwick J. BrittonThe University of Sydney

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Wozniak et al. (2006) studied this question.

synapsesocial.com/papers/6a79e3fe5968508e56f39cd5https://doi.org/10.4049/jimmunol.177.12.8684
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Immunology of Tuberculosis2001 · 2,163 citations
  2. 2Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation2003 · 1,658 citations
  3. 3TLR9 regulates Th1 responses and cooperates with TLR2 in mediating optimal resistance to <i>Mycobacterium tuberculosis</i>2005 · 592 citations
  4. 4IL-17 Stimulates the Production and Expression of Proinflammatory Cytokines, IL-β and TNF-α, by Human Macrophages1998 · 870 citations
  5. 5Characterization of IL-12 receptor beta1 chain (IL-12Rbeta1)-deficient mice: IL-12Rbeta1 is an essential component of the functional mouse IL-12 receptor1997 · 206 citations