Key result
ATF3 mRNA levels were significantly higher in aldosterone-producing adenomas compared to cortisol-producing adenomas (P = 0.0053), suggesting its involvement in aldosterone biosynthesis.
p-value: p=0.0053
ATF3 is highly expressed in aldosterone-producing adenomas and its expression is regulated by angiotensin-II via SRC and CaMKII pathways, suggesting a role in aldosterone biosynthesis.
ATF3 may regulate aldosterone production in adenomas; hypothesis-generating in animal models and requires human validation before clinical consideration.
The activating transcription factor 3 (ATF3) is a member of the cAMP-responsive element-binding (CREB) protein family of transcription factors. ATF3 is expressed in H295R human adrenocortical carcinoma cells and considered a rapid-responder gene to angiotensin-II stimulation. However, the functions of ATF3 in human adrenocortical tissues have remained unknown. In this study, we analyzed the localization and possible regulatory mechanisms of ATF3 in human adrenocortical cells and tissues. The expression levels of ATF3 mRNA were analyzed in 66 aldosterone-producing adenomas (APA) and 14 cortisol-producing adenomas (CPA) using real-time RT-PCR. To localize the ATF3 protein, we performed immunohistochemical analysis in 20 non-pathological adrenal glands, 9 adrenal glands with idiopathic hyperaldosteronism (IHA), 20 APA, and 5 CPA using a mouse monoclonal antibody against human ATF3. We showed that ATF3 mRNA levels were higher in APA compared to CPA (P = 0.0053). ATF3 was immunolocalized to the zona glomerulosa of non-pathological adrenal glands and adrenal glands with IHA, and diffusely detected in the tumor cells of APA and CPA. Subsequently, H295R cells were treated for 6 h with each inhibitor of Src kinase (SRC), PKC, JAK2, and calcium-dependent calmodulin kinase-II (CaMKII) in the presence or absence of angiotensin-II. The expression levels of ATF3 mRNA were increased by angiotensin-II (about 3.5-fold induction), but the magnitude of the induction was significantly decreased in the presence of an inhibitor for SRC (PP2) or CaMKII (KN93). These results suggest that ATF3 is a downstream target of SRC and CaMKII signaling, and may be involved in adrenocortical aldosterone synthesis.
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Felizola et al. (2014) studied Adrenocortical adenomas (n=80). Aldosterone-producing adenomas (APA) vs. Cortisol-producing adenomas (CPA) was evaluated on ATF3 mRNA expression levels (p=0.0053). ATF3 mRNA levels were significantly higher in aldosterone-producing adenomas compared to cortisol-producing adenomas (P = 0.0053), suggesting its involvement in aldosterone biosynthesis.
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