Population
Cardiac myocytes and fibroblasts isolated from knock-in mice expressing a FRET-based cGMP indicator
Design
Preclinical
Key result
NO-induced cGMP formed in cardiac fibroblasts enters cardiac myocytes via gap junctions, a process that is abolished by gap junction inhibitors.
Authors
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Does not support clinical translation; leaves open in vivo relevance of fibroblast-myocyte cGMP transfer.
NO-induced cGMP formed in cardiac fibroblasts enters cardiac myocytes via gap junctions, acting as an intercellular signaling molecule.
Menges et al. (2019) studied this question. NO-GC-activating substances was evaluated on cGMP signals in cardiac myocytes and fibroblasts. NO-induced cGMP formed in cardiac fibroblasts enters cardiac myocytes via gap junctions, a process that is abolished by gap junction inhibitors.
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