Diabetic macular edema (DME), which causes retinal thickening, is a main cause of visual impairment in patients with diabetes (1,2). The important pathophysiology of DME is the loss of retinal capillary pericytes, resulting in increased vascular permeability of the blood-retinal barrier (BRB) (3). However, there is only one report about the active transport of the BRB in patients with DME (4). The aim of this study was to evaluate the active transport of the BRB in patients with clinically significant diabetic macular edema (CSME) (5) in type 2 diabetes using differential vitreous fluorophotometry (DVF). We studied six eyes of six patients with …
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Mori et al. (2002) studied this question.
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