The Effect of Angiotensin Ii on the Sensitivity of the Parotid Gland of the Sheep to Aldosterone

Summary Aldosterone was infused intravenously into sodium‐replete or sodium‐deplete adrenalectomized sheep until the fall of salivary Na/K ratio had stabilized. Then valine‐5‐angiotensin II was infused simultaneously into the carotid artery supplying the isolated parotid gland. Angiotensin did not cause a further full in the salivary Na/K ratio. The results suggest that the factor which sensitizes the parotid gland to the action of aldosterone in sodium depletion is not angiotemin II.