Cardiac fibroadipocyte progenitors express desmosome proteins and differentiate into adipocytes upon desmoplakin gene deletion via a Wnt-dependent mechanism, expanding the cellular spectrum of arrhythmogenic cardiomyopathy.
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Does not inform clinical decisions in arrhythmogenic cardiomyopathy; extends fibroadipocyte progenitor origins in desmosomal models.
Lombardi et al. (2016) studied this question.
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