Why the study?
Does doxorubicin induce detrimental changes in myocardial energy metabolism and signaling systems?
Does doxorubicin induce detrimental changes in myocardial energy metabolism and signaling systems?
Doxorubicin-induced cardiotoxicity is driven in part by early, detrimental alterations in myocardial energy-transferring and signaling systems, including creatine kinase and AMPK.
Doxorubicin and other anthracyclines are among the most potent chemotherapeutic drugs for the treatment of acute leukaemia, lymphomas and different types of solid tumours such as breast, liver and lung cancers. Their clinical use is, however, limited by the risk of severe cardiotoxicity, which can lead to irreversible congestive heart failure. There is increasing evidence that essential components of myocardial energy metabolism are among the highly sensitive and early targets of doxorubicin-induced damage. Here we review doxorubicin-induced detrimental changes in cardiac energetics, with an emphasis on the emerging importance of defects in energy-transferring and -signalling systems, like creatine kinase and AMP-activated protein kinase.
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Tokarska-Schlattner et al. (2006) studied this question.
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