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June 1, 1998The Journal of Clinical Endocrinology & Metabolism

Insulin Stimulates Testosterone Biosynthesis by Human Thecal Cells from Women with Polycystic Ovary Syndrome by Activating Its Own Receptor and Using Inositolglycan Mediators as the Signal Transduction System1

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Authors

JNJohn E. NestlerEuropean Gravitational ObservatoryDJDaniela JakubowiczWolfson Medical CenterAVAı́da Falcón de VargasHospital Vargas

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Cite This Study

Nestler et al. (1998) studied this question.

synapsesocial.com/papers/6a7f73f462857677e1ca77b7https://doi.org/10.1210/jcem.83.6.4886
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Changes in Theca and Granulosa Cell Function in Antral Follicles Developing during Pregnancy in the Rat: Gonadotropin Receptors, Cyclic AMP and Estradiol-17β1979 · 75 citations
  2. 2Metabolic regulation of androgen production by human thecal cells in vitro1995 · 172 citations
  3. 3Insulin action in human granulosa cells from normal and polycystic ovaries is mediated by the insulin receptor and not the type-I insulin-like growth factor receptor.1995 · 202 citations
  4. 4Insulin-Signaling Mechanisms: Lessons From the Old Testament of Glycogen Metabolism and the New Testament of Molecular Biology1988 · 97 citations
  5. 5Insulin Mediators Are the Signal Transduction System Responsible for Insulin’s Actions on Human Placental Steroidogenesis*1991 · 50 citations