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March 23, 2004BloodOpen Access

GILZ, a new target for the transcription factor FoxO3, protects T lymphocytes from interleukin-2 withdrawal–induced apoptosis

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Authors

MAMarie-Liesse Asselin-LabatThe University of MelbourneMDMuriel D. DavidUniversité Claude Bernard Lyon 1ABArmelle Biola‐VidammentInserm

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Asselin-Labat et al. (2004) studied this question.

synapsesocial.com/papers/6a7faf3ac23602c964406c8ahttps://doi.org/10.1182/blood-2003-12-4295
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1FOXO Transcription Factors as Regulators of Immune Homeostasis: Molecules to Die for?2003 · 156 citations
  2. 2The Many Forks in FOXO's Road2003 · 431 citations
  3. 3FOXO transcription factors directly activate <i>bim</i> gene expression and promote apoptosis in sympathetic neurons2003 · 645 citations
  4. 4Interleukin-2 Inhibits Glucocorticoid Receptor Transcriptional Activity through a Mechanism Involving STAT5 (Signal Transducer and Activator of Transcription 5) but Not AP-12001 · 67 citations