Macroscopic pathology of Eisenmenger syndrome demonstrates the structural consequences of long-standing shunting, including right ventricular hypertrophy and pulmonary artery atherosclerosis and thrombosis.
Reaffirms pulmonary vascular sequelae in Eisenmenger syndrome; leaves open optimal antithrombotic strategies pending prospective data.
Figure. A and B - Gross examination of the heart. A - Opened right cardiac chambers. The asterisk marks the atrial septal defect located at the fossa ovalis (so-called secundum defect); B - The right ventricle hypertrophy and pulmonary artery trunk (PT) dilation with atherosclerotic plaques; C and D - Macroscopic findings of the lungs; C - The dilation and atherosclerosis of the pulmonary artery (PA) (arrowhead points to a plaque) and its branches; D - PA thrombosis (white arrowhead). TV = tricuspid valve.
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Campos et al. (2017) studied this question.
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