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March 1, 1998Journal of Biological ChemistryOpen Access

A Dominant Negative Isoform of the Long QT Syndrome 1 Gene Product

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Population

Human adult ventricle tissue and COS-7 cells

Comparison

Expression of KvLQT1 isoform 2 vs Expression of KvLQT1 isoform 1 alone or with IsK

Design

Preclinical

Authors

SDSophie DemolombeIBIsabelle BaróYPYann Péréon

Discussion

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Overview

Supports isoform-specific IKs regulation in animal myocytes; leaves open relevance to human LQT1.

Structured PICO

P
Population
Human adult ventricle tissue and COS-7 cells
I
Intervention
Expression of KvLQT1 isoform 2 (alone or coexpressed with isoform 1 and/or IsK)
C
Comparator
Expression of KvLQT1 isoform 1 alone or with IsK
O
Outcome
K+ current amplitude and activation kineticssurrogate

The identification of a dominant negative isoform of KvLQT1 provides mechanistic insight into the composition and regulation of the delayed rectifier K+ current in cardiac myocytes, relevant to long QT syndrome 1.

Cite This Study

Demolombe et al. (1998) studied this question.

synapsesocial.com/papers/6a815b8526f0d7dff79cd5behttps://doi.org/10.1074/jbc.273.12.6837
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