Episodes of hyperkalemia associated with severe muscle weakness dominated the clinical course of a patient with salt-losing nephropathy. The evaluation of urinary excretory patterns of electrolytes under varying intakes of sodium suggested that a reduction of glomerular filtration below a critically low rate was the main factor responsible for renal retention of potassium with resulting hyperkalemia.
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Mordecai M. Popovtzer (1973) studied this question.