CLINICAL,1 angiographic2 and experimental observations3 indicate that the larger arteries of the brain, like other arteries of equivalent size, are capable of active vasospasm that may have serious or even fatal consequences.Because of the important clinical implications of cerebral vasospasm an analysis of pertinent information bearing on the subject seems both timely and appropriate, especially since a climate of doubt still appears to surround this concept. Perhaps this doubt stems in part from the days when the concept of cerebral vasospasm leaned more heavily on theory than on radiologic and surgical studies of the behavior of major cerebral arteries. . . .
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James L. Pool (1958) studied this question.
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