Guillain-Barré Syndrome with Slow Progressive Onset and Persistent Elevation of Spinal Fluid Protein

C LASSIFICATION of the Guillain-Barre syndrome has been the subject of much discussion and review (1-11). The diversity of the clinical manifestations has led some clinicians to establish criteria that would more precisely delimit the syn-drome (1). Others take issue with this re-strictive tendency, believing that it excludes certain clinical manifestations such as pro-found sensory loss, relapses, and ataxia that should be considered part of the syndrome (2-5, 12). This attempt at restrictive classi-fication of the syndrome would be securely based if the etiologic mechanism(s) were understood. Despite the widely accepted parainfectious etiologic component, it is