Periodontitis is one of the most prevalent chronic diseases, affecting more than 743 million people worldwide (Jin et al., 2016). It can be defined as an inflammatory disease induced by oral polymicrobial community dysbiosis. The disproportionate host-response to pathogen penetration results in a continuous release of proinflammatory mediators and chronic inflammation, characterized by progressive and painless destruction of the tooth supporting apparatus.
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Brun et al. (2019) studied this question.
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