Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
June 29, 2009Arthritis & Rheumatism

The CGGGG insertion/deletion polymorphism of the IRF5 promoter is a strong risk factor for primary Sjögren's syndrome

View Full Paper
Ask AI
Bookmark
Share

Authors

CMCorinne Miceli‐RichardDélégation Paris 5NGNicolas GestermannUniversity Hospital of LausanneMIMarc IttahUniversité Paris-Sud

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Miceli‐Richard et al. (2009) studied this question.

synapsesocial.com/papers/6a83dcab07bf35307a996fbahttps://doi.org/10.1002/art.24662
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Failure to Detect Antiviral Activity in Serum and Plasma of Healthy Individuals Displaying High Activity in ELISA for IFNWOLFRAM alpha beta and IFNral1999 · 48 citations
  2. 2Association of an IRF5 gene functional polymorphism with Sjögren's syndrome2007 · 181 citations
  3. 3Comprehensive evaluation of the genetic variants of interferon regulatory factor 5 (IRF5) reveals a novel 5 bp length polymorphism as strong risk factor for systemic lupus erythematosus2007 · 192 citations
  4. 4Activation of IFN pathways and plasmacytoid dendritic cell recruitment in target organs of primary Sjögren’s syndrome2006 · 597 citations
  5. 5Interferon-inducible gene expression signature in peripheral blood cells of patients with severe lupus2003 · 2,283 citations