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In chronically sympathectomized cats Vidian nerve stimulation evoked vasodilatation concomitant with an increase of VIP concentration in venous plasma effluent from the nasal mucosa. the VIP increase and the vasodilatation was not affected by atropine administration, but abolished by hexamethonium. It is suggested that neuronal VIP may be the mediator of the atropine-resistant vasodilatation.
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Uddman et al. (1981) studied this question.
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